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Bypassing copper’s ATOX1-mediated copper toxicity.
Cupric ion (Cu(2+)) is toxic to a variety of organisms but its toxicity to mammalian cells is low compared to other transition metals and in some cases, it can even be beneficial. The transcription factor, Atox1, likely contributes to Cu(2+) detoxification in mammals by regulating expression of genes that confer resistance to the metal. The Cu(2+) sensing, Cu(2+)-AT